Tuesday, March 29, 2011
More practical advice for radiation exposure
Thursday, March 24, 2011
Baking Soda for Radiation?

Tuesday, March 22, 2011
How much potassium iodide should I take?

Exposures greater than 5 cGy:
Birth through 1 mo. - 16 mg.
1 mo. through 3 yrs. - 32 mg.
3 yrs. through 18 yrs. - 65 mg. (adolescents > 150 lbs. use adult dose)
Exposures greater than 10 cGy:
18 yrs. through 40 yrs. - 130 mg.
Exposures greater than 500 cGy:
Adults over 40 yrs. - 130 mg.
Potassium iodide protects for approximately 24 hours, and should be dosed daily until the risk no longer exists. First dose priority should be given to pregnant females and neonates, but a repeat dose should be given only if there is not proper evacuation and sheltering for them.
Persons with known iodine sensitivity should avoid potassium iodide, as should individuals with dermtitis herpetiformis, hypocomplementemic vaculitis, extremely rare conditions associated with an increased risk of ioding hypersensitivity. Individuals with multinodular goiter, Graves' disease, and autoimmune thyroiditis should be treated with caution -- especially if dosing extends beyond a few days.
Do not take ACE inhibitor blood pressure medication such as captropril, enalapril or lisinapril, or potassium sparing diuretics with the potassium iodide or the levels of potassium may go too high causing heart arrhythmias.
DO NOT take this amount of potassium iodide as a preventative measure. It will not serve the body and may harm it without the radiation to replace. If you want to use iodine and/or potassium iodide to reduce the affects of future exposure, take 500 mcg to 1 mg a day (MUCH less than the recommended dose for exposure) for at least 2 weeks to make sure the body has adequate iodine for all of its needs. You can continue to take 250 - 500 mcg. after the first two weeks to keep you level stable.
Until we meet again,
Dr. Judi
Friday, March 18, 2011
What to do for radiation exposure

Potassium iodide is going to be helpful, but for larger exposures it is not enough. First of all, the potassium iodide will help protect the thyroid, which is very susceptible to radiation, which can cause thyroid cancer. However, if you have low iodine levels in your body as many Americans do (in testing levels in my own patients up to 75% have low levels), just starting potassium iodide will not give you enough iodine to protect you. It takes several weeks on iodine supplements for the levels to rise to adequate levels to protect your thyroid. Iodine is also important for your breasts and other organs. So you may want to ask your doctor to check your blood iodine levels. If exposure is imminent and you don't have time to check your levels, a week or two of exta iodine will not hurt you.
Iodine and iodide work differently in different organs in the body. A better supplement would be one that uses both iodide and iodine, such as Iodoral or Lugol's solution.
The potassium in potassium iodide is also supposed to protect the body from cesium. However, you would have to overload on potassium iodide to get enough potassium, and that wouldn't be healthy for your thyroid. So it would be important to take extra potassium as well as the potassium iodide. Most supplements only contain 99 mg of potassium, so during radiation exposure you should take at least 5 a day (500 mg). If your kidneys are normal your body will handle that much well. If you have kidney failure your body already stores potassium.
Multiple trace minerals also have very important functions in the body. Taking adequate amounts of trace minerals will reduce the amount of radioactive minerals taking hold in the body.
Potassium and potassium iodide/iodine are not enough to protect from the heavier metals such as uranium and strontium90. Strontium90 and other heavy metals can be removed by EDTA chelation (IV if acute exposure), but the uranium molecule is too large for EDTA. By taking a humic/fulvic acid supplement, the uranium molecule is reduced and more easily removed from the body.
A good detox formula to assist the glutathione pathway used by the cells to remove toxins includes:
Alpha Lipoic Acid 100-300 mg.
N-acetyl cysteine 500 mg three times a day
Selenium 200 mcg
Vitamin C 4,000-6,000 mg.
Vitamin E (preferably mixed toxopherols) 800-1200 IU
Anti-oxidants to reduce the damage caused by toxins
Taking 12-15 capsules of chorella a day will also help remove heavy metals.
I would love to say that I have all of these supplements available on my www.springtreehealth.com website, but alas, as yet I do not. But they are all available on the internet and many in health food stores. Our SuperMulti Plus does have adequate amounts of trace minerals and selenium, along with good levels of vitamin E (tocotrienols) and anti-oxidants. If you are taking or needing the Glucose Balance, it also contains Alpha Lipoic Acid.
We all pray that there will be no need for radiation exposure treatment. But there are already many toxins and even radiation in the environment, and utlizing some of this may be important for many of us, whether the Japanese reactor has a meltdown or not.
Until we meet again,
Dr. Judi
Wednesday, September 29, 2010
Dr. Judi's Dream Team
Friday, September 24, 2010
How are Environmental Toxins Affecting Our Children?
TOXIC AMERICA:
http://www.fliqz.com/aspx/permalinkblank.aspx?vid=da7b1310d17940cd9785f79a52464b4d
Until we meet again,
Dr. Judi
Sunday, September 12, 2010
Are Statins Beneficial in Preventing Heart Disease?

If you are taking a statin drug to prevent heart disease even though your cholesterol levels may be normal, think again.
The following is quoted from Family Practice News, Volume 40, Issue 12, Pages 10-11 (July 2010):
Studies Dispute Statins Benefits for Prevention
The only large clinical trial to find that statins reduce mortality in patients taking them for primary prevention—a group that comprises 75% of statin users—was so seriously flawed that its conclusions were termed “clinically inconsistent” and invalid by one group of researchers and “extreme and exaggerated” by another, according to separate reports.
In the first critique of the 2008 JUPITER (Justification for the Use of Statins in Primary Prevention) trial, Dr. Michel de Lorgeril of Université Joseph Fourier and Centre National de la Recherche Scientifique, Grenoble, France, and his associates, analyzed the methods and findings of this only major study to claim that rosuvastatin produced a striking decrease in mortality in low-risk patients who have no evidence of coronary heart disease. These findings, which immediately provoked controversy, “have undoubtedly propelled many healthy persons without elevated cholesterol levels onto long-term statin treatment,” Dr. de Lorgeril and his colleagues said.
First among its major flaws, JUPITER was terminated early and apparently without proper justification according to the study's own protocol prespecifications. Early termination, a practice confined largely to industry-sponsored trials, tends to introduce biases that exaggerate the benefit and minimize the long-term harm of the treatment being assessed, according to the researchers. In this case, it also meant that the study was stopped just as mortality curves between the statin group and the control group had begun to converge, “suggesting that the borderline significant difference between groups may have disappeared” if follow-up had not been prematurely terminated, they wrote.
The main justification for terminating JUPITER was explained as an “unequivocal reduction in cardiovascular mortality” with statin therapy. Yet the JUPITER investigators did not include data on cardiovascular mortality in their published report. Readers would have to infer this result by extrapolating from data on a table. When examined closely, however, that data showed identical cardiovascular mortality between the treatment groups.
“Such a lack of effect on cardiovascular mortality [together] with a strong effect on nonfatal complications strongly suggests a bias in the data set and should have led to the continuation of the trial rather than to its premature ending,” Dr. de Lorgeril and his colleagues asserted.
Second, the ratio of fatal to nonfatal MI was so low as to be “incredible.” In particular, the case-fatality rate in the placebo group was only 8% when it would be expected to be about 50%, “a clinical inconsistency that suggests a major flaw in the study.” Moreover, the case-fatality rate with rosuvastatin was 29%, which implies that the “beneficial” drug actually tripled the case-fatality rate, they noted.
Third, JUPITER failed to explain why a strikingly high number of deaths—19 of 31 deaths in the rosuvastatin group and 25 of 37 deaths in the placebo group—were attributed to cardiovascular causes other than MI or stroke. Proponents of the JUPITER study argued after its publication that this referred to causes “such as aneurysm rupture.”
“Would this mean that in the same period of time there were 6 fatal infarctions and 25 fatal aneurysm ruptures in the placebo group? This is highly unlikely,” Dr. de Lorgeril and his associates said.
JUPITER also failed to report data on sudden cardiac deaths, which is surprising given that this is “the simplest and most reliable diagnosis in cardiology” and that it usually comprises 65%-70% of total cardiac mortality. “The way sudden cardiac death is reported—or not reported—may be a good indicator of the quality of the methods used in a trial,” they noted.
Equally important to these methodological flaws, the JUPITER trial involved several conflicts of interest.
“It was conducted by a sponsor with obvious commercial interests. Nine of 14 authors of the JUPITER article have financial ties to the sponsor. The principal investigator has a personal conflict of interest as a co-holder of the patent for the C-reactive protein test” that figured prominently in the rationale for statin therapy.
In addition, the monitoring board that made the decision to halt the trial early was chaired by an investigator who “has been and still is involved in many other industry-sponsored lipid-lowering trials,” Dr. de Lorgeril and colleagues said (Arch. Intern. Med. 2010;170:1,032-6).
“In conclusion, the results of the JUPITER trial are clinically inconsistent and therefore should not change medical practice or clinical guidelines.” Given that all 12 of the other large cholesterol-lowering trials conducted so far have failed to show any benefit from statins as primary prevention, it appears that “the presumed preventive effects of cholesterol-lowering drugs have been considerably exaggerated,” they said.
In the second study, Dr. Kausik K. Ray of the University of Cambridge (England) and his associates performed a meta-analysis of 11 randomized controlled trials that assessed the effects on all-cause mortality of statins versus a placebo or control therapies on all-cause mortality. They restricted their analysis to data on high-risk patients with no known cardiovascular disease and included previously unpublished data, “to provide the most robust information to date” on statins as primary prevention in this patient group.
The meta-analysis involved 65,229 men and women in predominantly Western populations, with approximately 244,000 person-years of follow-up. There were 2,793 deaths during an average of 4 years of follow-up.
All-cause mortality was not significantly different between patients taking statins and those taking placebo or control therapies. This suggests that “the all-cause mortality reduction of 20% reported in JUPITER is likely to be an extreme and exaggerated finding, as often occurs when trials are stopped early,” Dr. Ray and his colleagues said (Arch. Intern. Med. 2010;170;1024-31).
This meta-analysis shows that statin therapy as primary prevention in high-risk patients is less beneficial than is generally perceived, and it can be inferred to be even less helpful in low-risk patients.
One of Dr. de Lorgeril's associates served as an expert in litigation involving the pharmaceutical industry (not involving rosuvastatin). Dr. Ray and an associate reported financial ties to the majority of companies that market lipid-lowering agents; other associates reported ties to Pfizer Inc., Astra Zeneca, Bristol-Myers Squibb, and Merck & Co.
My Take
Both Critiques Are On-Target
These two critiques offer valuable insights and likely will reignite the long-simmering controversy over the use of statins as primary prevention of cardiovascular events.
Dr. de Lorgeril's critical analysis highlights several anomalies in the JUPITER trial data. In particular, early termination of the study allowed for inflated estimates of benefits, understated harms, allowed the findings to be published earlier (and hence used to advantage in marketing), and reduced the cost of the trial—which all significantly benefited the industry sponsor and a financially invested research team.
Tens of billions of dollars of revenue for the sponsor over the patent life of the drug were at stake in the JUPITER trial, as well as potentially millions of dollars in royalties for the principal investigator. And with three-quarters of statin users taking the drugs for primary prevention, enormous revenues are at stake.
The analysis by de Lorgeril et al. clearly demonstrates why research must be free of incentives to find a particular desired result.
Dr. Ray's report presents what is to date the cleanest and most comprehensive meta-analysis of pharmacological lipid lowering for prevention.
The results make it clear that for primary prevention in the short term, statins' benefits are very small. And in the long term, although sincere advocates on both sides will try to convince us otherwise, we really must admit that we do not know.
LEE A. GREEN, M.D., is in the department of family medicine at the University of Michigan Medical School, Ann Arbor. He reported no financial conflicts of interest. His comments appeared in an editorial (Arch. Intern. Med. 2010:170;1007-8).
From Archives of Internal Medicine
PII: S0300-7073(10)70742-8
doi:10.1016/S0300-7073(10)70742-8
Dr. Judi here: Statin medications have a lot of side effects which have an insidious onset and are hard to tell that the statin is causing them, including muscle pain and memory loss.
Other studies have shown that the best benefit from statins comes from those with high cholesterol and high cardiac c-reactive protein between the ages of 40-60. It is interesting to note that the elderly do not receive benefit from lowering their cholesterol. In fact, those with the lowest cholesterol have higher death rates than those with higher cholesterol. So when your doctor tells you that the statin drugs are the new wonder drug, look at benefits vs. the risks, do some research, and determine what is best for you.
Until we meet again,
Dr. Judi